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Since the high expression of TLR4 is closely linked with the development of cervical cancer but is not mediated by HPV, we reasoned that other pathogens, such as bacteria, may activate the TLR4 pathway to facilitate immune escape, which promotes the development of cervical lesions

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57,58 Accumulated DNA damage and cellular senescence are thought to be a common underlying mechanism linking infertility, cancer, cardiovascular disease, and other chronic diseases