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Elevated levels of reactive oxygen species (ROS) contribute to genomic instability and promote tumor initiation and progression in CSCs, while in NDDs such as Alzheimer’s and Parkinson’s disease, OS accelerates neuronal death and impairs cellular repair mechanisms
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SENP3 aggravates renal tubular epithelial cell apoptosis in lipopolysaccharide-induced acute kidney injury via deSUMOylation of Drp1
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