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A Nature Communications paper describes MOTS-c as a mitochondrial-encoded peptide and reports that exercise increased endogenous MOTS-c expression in human skeletal muscle and circulation
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Strength:5 mg
Chakrabarty et al., 2005), it may be therapeutic to reduce the activation of a glutamate receptor whose excessive activation can lead to neurotoxic sequelae (Hardingham & Bading, 2010)
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